Owen Davis | 19 May '21
Mercury can enter the body through dental amalgam, pesticides/fertilisers and seafood. After it enters the mouth, it is absorbed by any mucosal tissue (often the gums, or lungs where it is inhaled). Mercury can also enter the body through the eyes - particularly by way of conjunctivitis eye drops which contain the preservative, mercuric acetate. Upon absorption into the mucosal tissue, mercury can enter the bloodstream. Mercury is able to do this due to the high affinity for haemoglobin and sulphur binding proteins (proteins and cholesterol).
Hitching a ride on the 'blood-highway', mercury can go anywhere in the body - often ending up in vital organs, like the kidneys, liver and brain. It can also find its way into bone. When mercury ends up in vital organs, it will often induce a secondary immune response, which is the catalyst for a new chronic disease. Mercury can also work with bacteria, 'bat cave' viruses, parasites, cancer, tumours, fungi, yeasts, moulds, solvents, pesticides and other metals, such as gold, titanium and nickel (stainless steel crowns and dentures) to further weaken the immune system. And herein lies the danger of mercury.
So, how does mercury affect the body and what are some of the conditions associated with mercury poisoning?
Mercury toxicity is associated with over 250 symptoms, the most common of which are fatigue, depression and kidney dysfunction. There are, however, much more sinister conditions that arise because of mercury toxicity…
Brain / Nervous System / Myelin Sheath
Unlike other heavy metals, mercury can pass through the blood brain barrier.
Peripheral neuropathy may occur when mercury causes degeneration of the myelin sheath around nerves. The nerves begin to misfire
signals to the brain, which is caused by inefficient neuron energy utilisation.
Neurotoxins like mercury, are transported throughout the body attached to protein components of lipoproteins, and therefore require cholesterol for their transport and elimination. These neurotoxins also have a strong affinity for lipoidal tissue of the nervous system and brain. A rise in cholesterol levels and triglycerides in response to neurotoxins protects by preventing permanent attachment of the neurotoxin to the nerve and brain cells. Symptoms of neurotoxicity are most likely to occur when the cholesterol is lowered suddenly or when the affected client goes on a low-fat, low-cholesterol, low-protein diet.
A correct cholesterol response is fundamental to move mercury and other neurotoxins to sites where they can be excreted. A Danish study of 50,318 users of statin (cholesterol-lowering) drugs revealed a higher risk of peripheral neuropathy related to the percentage of drop in total cholesterol. In other words, lowering cholesterol increases risk of reactivity to nerve toxins resulting in pain, paraesthesia, numbness and demyelinating effects. Six additional studies since 1994 have indicated the same rise in poly peripheral neuropathy symptoms for users of statin drugs, supporting our clinical findings that low cholesterol levels in the presence of a potent neurotoxin such as mercury found in amalgam fillings or any other source, is a recipe for disaster.
Hashimotos and Graves' Disease
- mercury seeps into the jaw which has an embryological connection with the thyroid, blocking T4 to T3 and then usually resulting in an elevated TSH. In some cases, treating the thyroid may overlook mercury's presence. Always note dental history.
Do not overdose on Iodine (Potassium Iodide or Lugol's). When protein is low and mercury is present, too much iodine can create thyroid disorders by blocking receptor sites. Breast Cancer and Iodine deficiency have also been linked, but what if mercury is really behind the scene? Always look for Mercury's footprints in women who are of child bearing age, low iron or fast losing protein through menopause.
Kidney / Bladder
Prostate / Testes
Ovaries / Uterus / Womb
Muscle, Bone and Joints
Spleen and Bone Marrow
Toxicol In Vitro 2003 Aug;17(4):385-95. doi: 10.1016/s0887-2333(03)00040-7.
Cytotoxicity of inorganic mercury in murine T and B lymphoma cell lines: involvement of reactive oxygen species, Ca(2+) homeostasis, and cytokine gene expression
Environ Res 1998 May;77(2):149-59. doi: 10.1006/enrs.1997.3816.
Low-level methylmercury exposure causes human T-cells to undergo apoptosis: evidence of mitochondrial dysfunction
Often those who suffer from iron deficiency (perhaps due to a poorly formulated vegan or vegetarian diet), anaemia, diabetes, hormone imbalances (menopausal women or men who have low testosterone), sarcopenia (poor muscle tone), osteoporosis and osteopenia. These people are stereotypically more at risk of mercury poisoning. This is because their conditions often leave them immunocompromised. When an immunocompromised individual, (especially those who have the above medical conditions) are exposed to mercury, the mercury poisoning will often induce a secondary immune response, which acts as a catalyst for another autoimmune disease or cancer risk.